Vascular Medicine Conference-CVC 2026

Vasculitis represents a collection of disorders marked by inflammation within blood vessels, which can result in narrowing, weakening, or even rupture of these vessels. The implications of this process depend greatly on the size and anatomical location of the affected vessels, with potential involvement of organs such as the skin, kidneys, nervous system, and heart. Clinical presentations are highly variable, ranging from non-specific symptoms like fatigue, fever, and unintended weight loss to more targeted manifestations such as rashes, renal impairment, or neurological deficits.

Etiologically, vasculitis may arise from autoimmune mechanisms—where the immune system erroneously targets vascular structures—as well as infectious agents, adverse drug reactions, or remain idiopathic when no clear cause is identified. Prompt diagnosis is essential; delays can result in irreversible organ damage or, in severe cases, life-threatening complications.

Management strategies are primarily aimed at mitigating inflammation, preserving organ function, and addressing any underlying etiologies. Corticosteroids and various immunosuppressive agents form the cornerstone of therapy, supplemented by supportive measures and vigilant monitoring for potential complications. When recognized early and managed appropriately, patients may achieve disease control, reduce the frequency and severity of relapses, and maintain satisfactory vascular and organ health over the long term.